· Varieties of essential medicines
· Self-produced raw materials
· Quickly reduce ALT, protect liver and liver

Specification: 1.5mg
Drug category: Western medicine
National Health Insurance: Category A

Indications: Clinically used for chronic persistent hepatitis with elevated ALT, and can also be used for elevated ALT caused by chemical poisons and drugs.

Pharmacology and toxicology:
This product is an enzyme-lowering drug created in my country for the treatment of hepatitis, and is an intermediate in the synthesis of Schisandra C.

Oral administration of this product 150~200MG/KG to mice can reduce the liver damage and the elevation of alanine aminotransferase (ALT) caused by carbon tetrachloride. It has an inhibitory effect on the lipid peroxidation of liver microsomes caused by carbon tetrachloride and the conversion of carbon tetrachloride into carbon monoxide, and reduces the consumption of reduced coenzyme I and oxygen during carbon tetrachloride metabolism, thereby protecting liver cells. Structure and function of biofilms.

This product can also reduce the elevation of hepatic ALT induced by prednisone, and can promote liver regeneration in mice with partial hepatectomy. The enzyme-lowering effect of this product does not directly inhibit the activity of serum and liver ALT, nor does it accelerate the inactivation of ALT in the blood, which may be a reflection of the reduction of liver tissue damage. This product has obvious induction effect on cytochrome P450 enzyme activity, thereby enhancing the detoxification ability of carbon tetrachloride and some carcinogens. It can improve protein metabolism in some hepatitis patients, increase albumin and decrease globulin. It has no negative effect on HBSAG and HBEAG, nor can it shrink the enlarged liver and spleen.